References

Hirsch AT, Hartman L, Town RJ, Virnig BA. National health care costs of peripheral arterial disease in the Medicare population. Vasc Med. 2008; 13:(3)209-15 https://doi.org/10.1177/1358863X08089277

Becker F, Robert-Ebadi H, Ricco JB Chapter I: Definitions, epidemiology, clinical presentation and prognosis. Eur J Vasc Endovasc Surg. 2011; 42:S4-S12 https://doi.org/10.1016/S1078-5884(11)60009-9

Murabito JM, Evans JC, Nieto K Prevalence and clinical correlates of peripheral arterial disease in the Framingham Offspring Study. Am Heart J. 2002; 143:(6)961-965 https://doi.org/10.1067/mhj.2002.122871

Ubbink DT, Vermeulen H. Spinal cord stimulation for non-reconstructable chronic critical leg ischaemia. Cochrane Database Syst Rev. 2013; 2013:(2) https://doi.org/10.1002/14651858.CD004001.pub3

Labropoulos N, Leon LR, Bhatti A Hemodynamic effects of intermittent pneumatic compression in patients with critical limb ischemia. J Vasc Surg. 2005; 42:(4)710-716 https://doi.org/10.1016/j.jvs.2005.05.051

Baer WS. The classic: The treatment of chronic osteomyelitis with the maggot (larva of the blow fly). 1931. Clin Orthop Relat Res. 2011; 469:(4)920-944 https://doi.org/10.1007/s11999-010-1416-3

Cazander G, Pritchard DI, Nigam Y Multiple actions of Lucilia sericata larvae in hard-to-heal wounds: larval secretions contain molecules that accelerate wound healing, reduce chronic inflammation and inhibit bacterial infection. Bioessays. 2013; 35:(12)1083-1092 https://doi.org/10.1002/bies.201300071

Sherman RA. Mechanisms of maggot-induced wound healing: what do we know, and where do we go from here?. eCAM. 2014; 2014 https://doi.org/10.1155/2014/592419

Nigam Y, Morgan C. Does maggot therapy promote wound healing? The clinical and cellular evidence. J Eur Acad Dermatol Venereol. 2016; 30:(5)776-782 https://doi.org/10.1111/jdv.13534

Sherman RA. Maggot therapy for foot and leg wounds. Int J Low Extrem Wounds. 2002; 1:(2)135-142 https://doi.org/10.1177/1534734602001002009

Maeda TM, Kimura CK, Takahashi KT, Ichimura KI. Increase in skin perfusion pressure after maggot debridement therapy for critical limb ischaemia. Clin Exp Dermatol. 2014; 39:(8)911-914 https://doi.org/10.1111/ced.12454

Nordstrom A, Hansson C, Karlstrom L. Larval therapy as a palliative treatment for severe arteriosclerotic gangrene on the feet. Clin Exp Dermatol. 2009; 34:(8)e683-e685 https://doi.org/10.1111/j.1365-2230.2009.03405.x

Igari K, Toyofuku T, Uchiyama H Maggot debridement therapy for peripheral arterial disease. Ann Vasc Dis. 2013; 6:(2)145-149 https://doi.org/10.3400/avd.oa.13-00036

Bexfield A, Bond AE, Morgan C Amino acid derivatives from Lucilia sericata excretions/secretions may contribute to the beneficial effects of maggot therapy via increased angiogenesis. Br J Dermatol. 2010; 162:(3)554-562 https://doi.org/10.1111/j.1365-2133.2009.09530.x

Zhang Z, Wang S, Diao Y Fatty acid extracts from Lucilia sericata larvae promote murine cutaneous wound healing by angiogenic activity. Lipids Health Dis. 2010; 9 https://doi.org/10.1186/1476-511X-9-24

van der Plas MJ, van Dissel JT, Nibbering PH. Maggot secretions skew monocyte-macrophage differentiation away from a pro-inflammatory to a pro-angiogenic type. PloS One. 2009; 4:(11) https://doi.org/10.1371/journal.pone.0008071

Malvezzi L, Castronuovo JJ, Swayne LC The correlation between three methods of skin perfusion pressure measurement: radionuclide washout, laser Doppler flow, and photoplethysmography. J Vasc Surg. 1992; 15:(5)823-829 https://doi.org/10.1067/mva.1992.37088

Dumville JC, Worthy G, Bland JM Larval therapy for leg ulcers (VenUS II): randomised controlled trial. BMJ. 2009; 338 https://doi.org/10.1136/bmj.b773

Opletalova K, Blaizot X, Mourgeon B Maggot therapy for wound debridement: a randomized multicenter trial. Arch Dermatol. 2012; 148:(4)432-438 https://doi.org/10.1001/archdermatol.2011.1895

Horobin AJ, Shakesheff KM, Pritchard DI. Maggots and wound healing: an investigation of the effects of secretions from Lucilia sericata larvae upon the migration of human dermal fibroblasts over a fibronectin-coated surface. Wound Repair Regen. 2005; 13:(4)422-433 https://doi.org/10.1111/j.1067-1927.2005.130410.x

Cazander G, Schreurs MW, Renwarin L Maggot excretions affect the human complement system. Wound Repair Regen. 2012; 20:(6)879-886 https://doi.org/10.1111/j.1524-475X.2012.00850.x

Pritchard DI, Cerovsky V, Nigam Y TIME management by medicinal larvae. Int Wound J. 2016; 13:(4)475-484 https://doi.org/10.1111/iwj.12457

Bazalinski D, Kózka M, Karnas M, Wiech P. Effectiveness of chronic wound debridement with the use of larvae of Lucilia sericata. J Clin Med. 2019; 8:(11) https://doi.org/10.3390/jcm8111845

Steenvoorde P, Jacobi CE, Van Doorn L, Oskam J. Maggot debridement therapy of infected ulcers: patient and wound factors influencing outcome – a study on 101 patients with 117 wounds. Ann R Coll Surg Engl. 2007; 89:(6)596-602 https://doi.org/10.1308/003588407X205404

Nishijima A, Gosho M, Yoshida R Effective wound bed preparation using maggot debridement therapy for patients with critical limb ischaemia. J Wound Care. 2017; 26:(8)483-489 https://doi.org/10.12968/jowc.2017.26.8.483

Maggot debridement therapy in critical limb ischaemia: a case study

01 December 2020

Abstract

Objective:

In critical limb ischaemia (CLI), first-line therapy is revascularisation, but alternative treatment options are needed in certain cases. Maggot debridement therapy (MDT) is historically considered to be contraindicated in ischaemic ulcers. Wound care in patients with CLI is becoming increasingly diverse with the development of novel revascularisation strategies; therefore, CLI now needs to be reconsidered as an indication for MDT.

Method:

We retrospectively reviewed five legs with CLI (five male, one female) treated with MDT between January 2013 and December 2017. Changes in skin perfusion pressure (SPP) around the ulcer before and after MDT were evaluated. One or two cycles of MDT were performed (eight in total). We also evaluated the proportion of necrotic tissue in the ulcer and the presence of exposed necrotic bone. The proportion of necrotic tissue in the ulcer was classified as NT 1+ (<25%), NT 2+ (25–50%), NT 3+ (50–75%) or NT 4+ (>75%).

Results:

When the proportion of necrotic tissue was >50%, with no exposed necrotic bone in the wound, an increase in SPP was observed after five (62.5%) of eight cycles of MDT. And with a proportion of necrotic tissue of <25% and/or exposed necrotic bone in the wound, a decrease in SPP was observed after three (37.5%) of eight cycles. Wound healing was accelerated in the presence of increased SPP.

Conclusion:

Effective MDT with increased SPP requires an ulcerative state of necrotic tissue grade > NT 3+, with no exposed necrotic bone.

The prevalence of peripheral arterial disease (PAD) involving the lower extremities is increasing worldwide as a result of ageing populations, continued cigarette smoking, the diabetes epidemic and increasing obesity rates.1 Critical limb ischaemia (CLI) is the most advanced form of PAD, and is defined as the presence of chronic ischaemic pain at rest, ulceration or gangrene attributable to objectively proven arterial occlusive disease.2 CLI is associated with a high risk of cardiovascular events, including major limb loss, stroke and death.3 There are a number of treatments available for CLI, including pharmacologic therapy, interventional procedures and surgery, as well as other additional treatments, such as hyperbaric oxygen therapy, spinal cord stimulation4 and intermittent pneumatic compression.5

Maggot debridement therapy (MDT) plays an important role in the management of hard-to-heal infected wounds and is used worldwide. MDT has a long history in the clinical setting and its effectiveness has been recognised since ancient times. In more modern times, Baron Dominique Jean Larrey, Surgeon-General in Napoleon's army, was the first to describe the benefits of MDT in writing. In 1931, William Baer, an orthopaedic surgeon at Johns Hopkins Hospital in Baltimore, reported the usefulness of MDT in patients with chronic osteomyelitis.6 Although the mechanisms underlying MDT are still not completely understood, laboratory studies have identified various effects of larval secretions/excretions.7 MDT is now a widely accepted medical practice that has three main actions: debridement, disinfection and stimulation of tissue growth.8 Moreover, clinical and cellular evidence for the effect of MDT on wound healing has recently been discussed in detail.9

Register now to continue reading

Thank you for visiting Journal of Wound Care's Silk Road Supplement and reading some of our peer-reviewed resources for healthcare professionals across Asia. To read more, please register today.